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The history of the traction device The device resulted as a medical discovery by a urologist in Spain, Dr. Eduardo de Diego. The device was soon tested on over 30 "small" sized men, and on average, these men were able to gain an average of 28% increase n size in just 3 months with the use of the new traction device for penis enlargement. A detailed analysis of customer testimonials and unsolicited testimonials to some of the larger penis enlargement companies resulted in that the average user of the penis traction device can gain .75 gains per month! This is truly remarkable if you look back at some of the older methods still being used today, such as pumps and surgery. This is because long term use of pumps can bring physical problems such as dependence on the use of the pump after a while to even get an erection, and the high rate of failure or problems with post cosmetic penis enlargement surgery. What is the best device? This area of the article discusses factors that can lead to best device for anyone interested in the benefits of its use. Some of the devices I have tried were quite painful, and only could be worn at most 30 minutes at a time. Do you think the top urologists and male health doctors are using these substandard devices? I think not. These lower quality devices have not been improved upon for better quality experience and have simply hit the market too soon. The higher quality devices on the market though have gone through hard testing to be sure the device will be comfortable and highly effective. To focus on these higher quality devices, there are a few things to look out for on a given device website. For one thing, look for clear clinical studies proving the effectiveness of the device. Since the traction device for penis enlargement has grown in popularity, there have been some new companies promoting a device without any solid medical evidence to back up their claims. Another thing is to choose a device that comes with extra parts for free. If it does not, then you will have spent a good sum of money on a low quality device that brings limited results. Extra adjustable bars will permit you to keep enlarging if you wish to do so. Choose a device that offers you a at least a 6 month guarantee, because if you do notice any problems, you have the comfort of th 180 day guarantee to get your money back. Another way to find a quality device is if the device site has good support. Some penis enlargement product websites have a forum where their members talk about what is working for them. If you can find a site with an active forum, then you can be sure to find good information and reviews of the device your checking out, among many other products. Personal product review websites also are supposed to offer unbiased, objective reviews on the top penis enlargement and male enhancement methods. Consult these product review sites carefully, as many are just out to make a fast buck for high commissions. What you really want is to see which products are the most highly reviewed and rated, so read carefully what each device offer you in terms of benefits, results, guarantee, and more for the price they are asking for. Overall though, if you desire a larger penis, you will find all you need with the modern penis enlargement methods, such as the traction device These methods were designed by medical health doctors and scientists to be safe, discreet, and offer quick permanent results. Whether you want to have better sex, a larger penis, increase sperm count, or all 3 of these, the natural penis enlargement device may provide an easy solution for you. Welcome to the path to penis enlargement, and good luck on your gains! penis elargement traction device easy enhancement free penis surgery way best enlagement exercise penis do penile enlargment pills work penis enhancement drug penis enlarement exercise guide to penis elargement vimax free penis enlargement technique

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The word 'Mother' is synonymous with care, love and all good things on the earth. However, smoking mothers can be inadvertently killing and maiming their children – born and unborn. Shocked? It's true. Smoking mothers are potentially gifting their children with lung cancer and many other similarly devastating diseases through second hand smoke. Pregnant women harm their unborn babies directly in the womb. Besides the above horrifying facts, here are more unsettling reasons to bear in mind when smoking. It will not only interfere with your health, it will also interfere with the health of your unborn child: 1. First of all, smoking makes it hard to conceive. Reports show that fertility in both men and women are heavily affected by smoking. In women, smoking interferes with the egg movement toward the fallopian tubes as well as the hormones that are produced during pregnancy; while in men, it lowers the semen count, and the motility of the sperm. In many cases smoking also affects the shape of the sperm making it incapable of penetrating the egg. 2. Children born of smoking parents potentially inherit a low fertility syndrome and suffer from a great number of infertility problems. 3. Smoking in many cases is the main cause for impotency in men as it interferes with the blood supply to the penis. 4. The baby in the womb of a pregnant smoking woman gets its nicotine addiction right from pre-birth as its blood imbibes the nicotine that the mother’s blood shares with it; hence it is more likely that when born they would become a smoker – and also contract all smoking related diseases. 5. In rare cases the placenta is directly affected by smoking causing a miscarriage of the foetus, or death in the womb. 6. Babies of smoking mothers will be most likely to be born premature and under weight. 7. Babies are also likely to have less developed vital organs than their counterparts (babies of non-smoking mothers) – there is a great likelihood that these babies have an undeveloped liver which will affect them throughout their lives. 8. The babies of pregnant smoking women have poor lung functions right from birth. 9. These babies are twice as prone to die from sudden-infant-death (SID) syndrome than the babies of non-smoking women. 10. The babies of those women who smoked 15-20 cigarettes a day are more likely to be sickly. Any one of the above causes should be sufficient enough to make you quit smoking – if not for yourself, for your little miracle that is being formed in your womb. Read the above lines again, and then once more – is that what you want to give to your most precious creation? Would you really be comfortable thinking that you are voluntarily and consistently harming your child even before their born? These are some questions that you should to ponder seriously. Get in touch with your doctor today and enlist his/her help to quitting smoking. You owe it to your child to be healthy and give them a healthy beginning to the start of their life. As a mother, you are the nurturing contact of the child – do not become the poison in your own child’s life. penis enlarement surgeries free penis enlargement exercise herbal penis enlargment pills pnis enlargement technique do penis enlagement pills work top penis enlargement pill free pnis enlargement video pnis enlargement excercises pnis enlargement program

Hypertension can wreak havoc on many body organs. Its effects on the heart are especially damaging. High blood pressure and heart disease are closely connected. Moderate to high blood pressure significantly increases the load on the left side of the heart. Arterioles are often diseased or constricted, which increases resistance to blood flow. When this happens, the heart has to work much more to pump blood into the arterial system including the aorta. When any muscle is overused, it tends to increase in size and bulk. The heart muscles react the same way. An enlarged heart is a sign of trouble. Heart enlargement can be detected in several ways. A chest x-ray can reveal it. So can an electrocardiogram or even a physical examination. In many high blood pressure patients, the walls of the left ventricle thicken. This increases the workload on the heart. Eventually, the heart falters and the left side of the heart no longer pumps blood adequately. When this happens, major organs and tissues of the body are denied sufficient blood supply. The affected person becomes lethargic and weak. Because of the higher pressure in the left ventricle, blood from the lungs cannot drain into the top compartment of the heart (the atrium). As a result, the lung tissues get congested which brings on bouts of breathlessness and coughs. The patient may have a dry cough or even frothy phlegm with blood stains in it. This is a very scary experience to the sufferer and onlookers. In an acute attack, it seems to the patient that he or she is choking to death. Breathlessness due to heart problems may be first noticed during prolonged physical exertion. If the condition has progressed further, breathlessness may occur even while resting. This is a serious situation and needs to be tacked immediately. If this damage worsens, the right side of the heart will be affected as well. When pressure builds up in the right atrium, it will be difficult for veins to drain blood into it. When the heart's condition deteriorates to this stage, the external jugular veins may become enlarged and more prominent. These veins are anyway close to the skin and may be visible even in healthy adults, but stress on the heart can increase their protrusion. There are several other symptoms of right side heart failure. They include an enlarged liver, swollen ankles and feet, loss of appetite, swollen abdomen and lower urine flow. High blood pressure and heart disease frequently have a cause and effect relationship. Making necessary lifestyle changes can help combat both. does vimax work penis enhancement procedure do penile enlargment pills really work guide to penis enlarement penis enhancement surgery picture penile enlargment supplement manual penis enlarement penis enargement forum pnis enlargement program

When you are tired and stressed it take its toll on your body and one of the first things to suffer is sexual health. Here we will outline not only some natural supplements for male sexual health, but also the natural foods you can eat to make you feel healthier, happier and more sexual. Your overall health affects sexual wellbeing! When looking at male sexual health it is not just a question of taking a few supplements and sex drive recovers. You must see male sexual health in terms of overall body health, this means not just taking supplements, but eating foods to reduce stress first, then adding supplements to enhance your overall mood. Let’s take foods first: These foods will increase dopamine a chemical in the brain that will increase sex drive so eat them! They are: Salmon, cottage cheese, steak and low fat yogurt Salmon (and other oily fish such as mackerel and herring) are packed with Omega 3, which increases blood flow around the body and to the genitals and is vital for fighting impotence. Cherries, raspberries, blackcurrants and aubergines are rich in anthocyanins an antioxidant that prevents fatty deposits on the walls of blood vessels keeping blood running smoothly. This as we have said above in relation to salmon is crucial in avoiding the symptoms of impotence. One mineral that is crucial to sex drive is Zinc, so make sure you get plenty of it from crab, multigrain bread, oysters, red meat and sardines. Low levels of iron can also cause fatigue and kill sex drive, so eat plenty of lean red meat, dark turkey meat, chicken, eggs and oily fish; all of these are a good source of iron. The above foods should be eaten in a diet that includes plenty of vegetables and fibre for overall energy. These foods act as great stress busters and as a by-product improve male sexual health. Also keep in mind to cut down or stop taking alcohol or smoking, which are known sex drive killers So how about some natural supplements to boost improve male sexual health? Here are three of the best L argentine This nutrient is highly important for peak make sexual health and is probably the BEST Supplement you can take for male sexual wellbeing. Unlike many other supposed sex drive boosters; this one is medically proven to enhance sex drive in healthy males. Current studies support the use of argentine supplements to ensure that nitric oxide secretion is sufficient to keep blood flowing to the penis. Nitric oxide insufficiency can stop the penis from becoming erect. A recent study showed an 80 percent improvement in the erectile function of men who took 2.8 grams of argentine a day for two weeks. Gingko Bilbao It enhances blood flow and functions as an anti-oxidant in the body and is renowned as a great all round supplement which has been known for centuries to increase sexual desire. Ginseng Korean Ginseng has been used in China as a sexual balancer and revitalizing tonic for thousands of years. It is stimulating and restorative and helps improve physical and mental energy, stamina, strength and alertness as an adaptogen, it also helps you to combat physical, emotional stress and fatigue. It has a normalizing effect on hormone imbalances and increases metabolic rate and improves blood flow to the genitals. We know stress and tiredness are sex drive killers so by your diet right and taking proven supplements to enhance your sexual mood, you will feel healthier and have a stronger sex drive real pennis enlargement penile enlargement program penis enlargement tip penis enlargement surgeon bottle vimax pills penis enlargment doctor free penis enlagement natural penis enlargment exercise pnis enlargement program

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth.